Abstract:
This study investigates the local stability properties of a virus dynamic model for HIV-I virus infection including saturation infection rate. The novel feature is that both the absorption effect and the cure rate are incorporated into the model with the intracellular time delay. This study was undertaken to present the local stability and how the basic reproduction number influences the local stability of the model. We included four significant and distinctive aspects in the proposed model compared to the usual HIV-I mathematical models available in the literature.